Oral Turinabol is suppressive to natural testosterone and should be used in conjunction with exogenous testosterone. Men who use Oral Turinabol without exogenous testosterone will risk a low testosterone condition. Such a condition can come with a host of possible symptoms ranging from physical, mental and sexually related. However, while physical related symptoms are unlikely when steroids are being used the others are a very real possibility.
Once the use of Oral Turinabol comes to an end natural testosterone production will begin again on its own. However, natural levels will still be very low and it will take a large amount of time to recover proper or healthy levels. For this reason most men are encouraged to implement a Post Cycle Therapy (PCT) plan once the use of anabolic steroids is discontinued. This will greatly speed up the recovery process and protect your lean tissue. Without a PCT plan it is possible for cortisol to become dominant for a period of time, destroy muscle tissue and promote fat gain. While a PCT plan will promote recovery, it will not return you to normal on its own. There is no PCT plan on earth that has this ability. However, a well planned PCT will speed up the process and ensure you have enough testosterone for proper bodily function while your levels continue to naturally rise.
There are a few important notes on natural recovery, the primary being that no low testosterone condition existed prior to anabolic steroid use. Further, natural recovery assumes no severe damage was done to the Hypothalamic-Pituitary-Testicular-Axis (HPTA) through improper anabolic steroid use. As a final note, women have no need to supplement with exogenous testosterone when using Oral Turinabol.
During the two 'off' weeks, an ECA stack can be used as required. ECA will not cause such a pronounced down regulation and desensitization of the receptors, certainly not to the extent of clen. Ephedrine has a short half life in contrast to clen which results in times throughout the day where the betas will partially recover from stimulation by adrenaline and nor-adrenaline. Potency is also much weaker that that of clen, as it is not a specific agonist. Ephedrine is also thought to increase the conversion of endogenous/exogenous T4 to T3 through the activation of deiodinase enzymes responsible for this process. This is important as clen is known to slow the rate of T4 to T3 conversion. As a side note, some bodybuilders will use T3 concurrently with the Clenbuterol/ECA cutting cycle (together with certain anabolic/androgenic steroids no doubt!) in an attempt to at least maintain plasma T3 levels.
Injectable steroids are injected into muscle tissue, not into the veins. They are slowly released from the muscles into the rest of the body, and may be detectable for months after last use. Injectable steroids can be oil-based or water-based. Injectable anabolic steroids which are oil-based have longer half-life than water-based steroids. Both steroid types have much longer half-lives than oral anabolic steroids. And this is proving to be a drawback for injectables as they have high probability of being detected in drug screening since their clearance times tend to be longer than orals. Athletes resolve this problem by using injectable testosterone early in the cycle then switch to orals when approaching the end of the cycle and drug testing is imminent.